AndyLL
DIS Veteran
- Joined
- Jan 10, 2008
- Messages
- 1,059
The chemical bond that connects two mono saccarides (like glucose and fructose) has very little to do with the way that those sugars are actually matabolized. It is a weak, easily broken chemical bond that is actually taken care of largely by stomach acid. I don't see how the fact that they are not connected could make a significant difference in their matabolism. I think it is more about the fact that the ratio of glucose to fructose is off in HCFS. Fructose is slightly different is structure to glucose an is more likley to be stored in the body, hence the problem with the high concentration of fructose. The way i read it, the results of the Princeton study don't necessarily point to the fact that they are bonded as significant, rather the ratio of fructose to glucose.
The Princeton study did not state the the bound/unbound was the issue but as a possiblity.
Their finding lead them to believe that HFCS does metabolize differently in the body.
The rats using HFCS had unique fat gain and increased triglycerides which also point to differences in how they metabolize.
The study was looking at if HFCS metablizes differently... not necessarily how.


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